Site Search



Bruce D. Teter, Ph.D.

Work Email Address:
bteter@ucla.edu

Mailing Address:
VA Greater Los Angeles Healthcare System, Sepulveda
Department of Medicine
16111 Plummer Street
MC 151
Sepulveda, CA 91343
UNITED STATES

Work Address:
Office
UCLA-VAMC
mc151
16111 Plummer St.
Sepulveda, CA 91343
UNITED STATES

Laboratory
SEPUL VAMC 7
Los Angeles, CA 90095
UNITED STATES

Cell-phone Number:
3104131759
Fax Number:
8188955835
Work Phone Number:
(818) 891-7711 x7481
8188917711x7645



Login to the Faculty Database

Department / Division Affiliations
Associate Professor, Geriatric Medicine
Member, Brain Research Institute
Adjunct Associate Professor, Department of Medicine

Bio:

Bruce Teter received a B.S. in marine biology and a minor in chemistry from Cal. State U., Long Beach in 1982. Dr. Teter received a Ph.D. degree in molecular biology from the University of Southern California, Los Angeles, in 1991. He then completed a postdoctoral fellowship in neurogerontology working with Dr. Caleb Finch at the Andrus Gerontology Center in the School of Gerontology at USC. In 1992, Dr. Teter was appointed Research Associate at the Andrus Gerontology Center. In 1996 he joined the lab of Dr. Greg Cole at UCLA and is currently Associate Professor in the Department of Medicine at UCLA, and Chief of the ApoE Research Laboratory, and Research Associate at GRECC, Veterans Administration, GLAHS.

Publications:

Teter, B. Life-span influences of apoE4 on CNS function. Invited Peer Commentary on: Schonheit, B., Glockner, F., and Ohm, T.G. (2006) Apolipoprotein E polymorphism and dendritic shape. . Neurobiology of Aging 2006; in press: .
Teter, B., Finch, C.E. Caliban's Inheritance: Genetics of Neuronal Aging. Trends in Neuroscience 2004; 10: 627-32.
Download
Teter B, Xu P-T, Gilbert JR, Roses AD, Galasko D, Cole GM Defective neuronal sprouting supported by human Apolipoprotein E4 represents a gain-of-deleterious function. J. Neurosci. Res 2002; (687): 331-336.
Download
Teter B, Ashford JW Neuroplasticity in Alzheimer's Disease. Journal of Neuroscience Research 2002; 70(Aging Brain and Alzheimer's Disease Special Issue): 402-437.
Download
Teter B, Raber J, Nathan B, Crutcher KA The presence of apoE4, not the absence of apoE3, contributes to AD pathology. J. Alzheimer's Disease 2002; (4): 155-163.